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Anatomically distant brain regions and the functional interaction between them. By comparing the members of these twin pairs with a series of normal control twin pairs, she will be able to assess whether the twins with the disorder show particular abnormalities, and if so whether they are also present in their well co-twins. Dr. Curtis seeks to identify the abnormalities associated with bipolar disorder, and also any abnormalities which are found in the healthy cotwin which might therefore predispose to the disorder and famotidine.
Individuals decreases the vascular resistance by 68% when normalized to each individual's maximal vasodilator capacity. The decrease in vascular resistance is of the same magnitude as in control subjects. To our knowledge, this is the first observation that -adrenergic tone of the leg skeletal muscle vascular bed is preserved in individuals without supraspinal sympathetic control. Upper leg blood flow is lower and upper leg vascular resistance is higher in SCI than in control subjects. This is in accordance with previous observations in an independent sample of SCI and control subjects, with a different technique used to measure leg blood flow.21 As previously discussed, 21 this is most likely due to structural a decrease in number of arterioles and capillaries and or a decrease in diameter of the resistance vessels ; as well as functional changes. Indeed, previous research, 6, 25 and the present observation that maximal vasodilation during postischemic hyperemia is reduced in SCI, supports the concept that structural changes in SCI contribute to the reduced baseline leg blood flow. Phentolamine-induced upper leg vasodilation in SCI, regardless the method of expressing the results relative or normalized ; , indicates preserved -adrenergic vascular tone in these individuals. The absolute upper leg blood flow in response to infusion of phentolamine was less in SCI as compared with control subjects. This is due to structural changes in the leg vascular bed of SCI, since this apparent difference disappeared when results were normalized to the individual maximal vasodilator capacity of the upper leg vascular bed. In control subjects, the vasodilator response to phentolamine was very similar to the response observed by others in a similar group of volunteers.2 Therefore, methodological problems are not likely to explain the lack of difference between the groups in vasodilator response to phentolamine in the present study. Since upper leg blood flow was lower in SCI than in control subjects, the same dose of phentolamine resulted in higher local plasma concentration of phentolamine in the infused leg in SCI compared with control subjects. However. Figure 4. MSA reduces ERa mRNA. MCF-7 A ; , MCF-7-LCC2 B ; , HEC1A C ; , and Ishikawa D ; cells 2 106 cells per plate ; were incubated with vehicle Veh ; , 108 mol L estradiol E2 ; , 107 mol L 4-hydroxytamoxifen Tam ; , or 10 Amol L MSA alone, or coincubated with estradiol and MSA or 4-hydroxytamoxifen and MSA for 2 h. ERa mRNA was measured by real-time RT-PCR as described in Materials and Methods. Expression was normalized to GAPDH. Columns, means; bars, FSD. * , P 0.05, compared with vehicle-incubated samples. c, P 0.05, compared with estradiol- or 4-hydroxytamoxifen-incubated samples. Mol Cancer Ther 2005; 4 8 ; . August 2005 and fexofenadine. Differing potencies as thyroid stimulators in various systems. Such variants of hCG have been described in trophoblastic disease 21 ; and in normal pregnancy sera 22 ; . Estradiol, which we and others have found to be present in greater concentrations in hyperemesis patients, provides a plausible link between hCG and the clinical finding of nausea and vomiting. Serum estradiol concentration may be related to hCG through the effect of hCG on steroidogenesis; hCG stimulates the corpus luteum to produce estradiol as well as progesterone 23 ; . hCG may also promote aromatization of placental steroids and stimulate fetal adrenal synthesis of dehydroepiandrosterone sulfate 24, 25 ; , both of which effects could lead to increased estradiol. The increased estradiol may, through its effect on the lactotrophs, be responsible for the greater concentration of PRL in hyperemesis subjects. It is not possible, however, to separate this effect from the acute effect of the phenothiazine antiemetic in raising serum PRL. The hypothesis showing the relationship between hCG, thyroid function, and estradiol is summarized in Fig. 4. Our findings support the hypothesis that hCG is the cause of the thyroid stimulation in hyperemesis gravidarum and is closely linked to the cause of the vomiting itself. Studies of thyroid function in early pregnancy should take into account the severity of vomiting, which occurs to some degree in the majority of pregnant women 26 ; . The syndrome of transient hyperthyroidism of hyperemesis gravidarum should be considered in any woman presenting with biochemical evidence of hyperthyroidism in early pregnancy. Acknowledgments. Him that will be there when he needs it? James Jaeger J.D., Elder Law Attorney, will open the workshop with Legal Issues and Financial Planning. David Mays M.D., Psychiatrist, will conduct on Meeting the Challenges of Individuals with Mental Illness As They Age. In the afternoon, Jan Greenberg, Ph.D., University of WI Waisman Center and School of Social Work, will present on The Future Role Of Siblings as a source of support to a brother or sister with mental illness. Donna Wrenn, NAMI WI Executive Director, will give a brief presentation on Developing An Individual Plan. Medicare Changes, Part D will be the day's last presentation and pseudoephedrine.
1. Fettes I. Migraine in the menopause. Neurology. 1999; 53 4 Suppl 1 ; : S29-33. 2. Launer LJ, Terwindt GM, Ferrari MD. The prevalence and characteristics of migraine in a population-based cohort. Neurology. 1999; 53: 537-542. Ratinahirana H, Darbois Y, Bousser M-G. Migraine and pregnancy: a prospective study in 703 women after delivery [Abstract]. Neurology. 1990; 40 suppl 1 ; : 437. 4. MacGregor A. Migraine associated with menstruation. Funct Neurol. 2000; 15 Suppl 3: 143-53. 5. Somerville BW. The role of estradiol withdrawal in the etiology of menstrual migraine. Neurology. 1972; 22: 355365. Somerville BW. The role of progesterone in menstrual migraine. Neurology. 1971; 21: 853. Magos AL, Zilkha KJ, Studd JWW. Treatment of menstrual migraine by oestradiol implants. Journal of Neurology, Neurosurgery, and Psychiatry. 1983; 46: 1044. Murray SC, Muse KN. Effective treatment of severe menstrual migraine headaches with gonadotropin-releasing hormone agonist and "add-back" therapy. Fertil Ster il. 1997; 67: 390-393. De Lignieres B, Vincens M, Mauvais-Jarvis P. Prevention of menstrual migraine by percutaneous oestradiol. British Medical Journal. 1986; 293: 1540. Predalier A, Vincent D, Beaulieu PH, et al. Correlation between oestradiol plasma level and therapeutic effect on menstrual migraine. In: Rose FC, ed. New advances in headache research, 4th ed. London: SmithGordon, 1994: 129-132. 11. Ryan, Sr. RE. Acontrolled study of the effect of oral contraceptives on migraine. Headache. 1978; 17: 250. D'Amico JF, Greendale GA, Lu JKH, Judd HL. Induction of hypothalamic opioid activity with transdermal estradiol administration in postmenopausal women. Fertil Steril. 1991; 55: 754-758. Calhoun A. Adjusting estradiol concentrations reduces headache frequency and severity in female migraineurs. Presented at the International Headache Congress, July 2001, New York, NY. 14. The Collaborative Group for the Study of Stroke in Young Women. JAMA. 1975; 231: 718-722. Petitti DB, Sidney S, Bernstein A, Wolf S, Quesenberry C, Ziel HK. Stroke in users of low-dose oral contraceptives. N Engl J Med. 1996; 335: 8-15. Fruzzetti F, Ricci C, Fioretti P. Haemostasis profile in smoking and nonsmoking women taking low-dose oral contraceptives. Contraception. 1994; 49: 579-592. Collins P, Shay J, Jiang C, et al. Nitric oxide accounts for dose dependent oestrogen-mediated coronary relaxation following acute oestrogen withdrawal. Circulation. 1994; 90: 1964-8. Zhang F, Ram JL, Standley PR, et al. 17- oestradiol attenuates voltage-dependent Ca currents in A7t5 vascular smooth muscle cell line. J Physiol. 1994; 266: C975-80. 19. Proudler A, Ahmed A, Crook D, et al. Hormone replacement therapy and serum angiotensin converting enzyme activity in postmenopausal women. Lancet. 1996; 346: 89-90. Rosano G, Patrizi R, Leonardo F, et al. Effects of estrogen replacement therapy on heart rate variability and heart rate in healthy postmenopausal women. J Cardiol. 1997; 80: 815-17.

Log in register now home page my times today's paper video most popular times topics wednesday, september 19, 2007 health guide world region business technology science health research fitness & nutrition money & policy views health guide sports opinion arts style travel jobs real estate autos health times health guide e estradiol - test estradiol - test overview alternative names how the test is performed how to prepare for the test how the test will feel why the test is performed normal values what abnormal results mean what the risks are special considerations news & features related topics ovarian cancer turner syndrome hypopituitarism an estradiol test measures the amount of estradiol in the blood and finasteride.
How generic and brand-name drugs are alike the federal drug administration fda ; regulates the drug industry and makes sure generic drugs work exactly the same way as their brand-name equivalents.

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32. Neish A, Williams A, Palmer H, Whitley M, Collins T. Functional analysis of the human vascular cell adhesion molecule 1 promoter. J Exp Med. 1992; 176: 15831593. Thanos D, Maniatis T. NF- B: a lesson in family values. Cell. 1995; 80: 529 Kliewer SA, Moore JT, Wade L, Staudinger JL, Watson MA, Jones SA, McKee DD, Oliver BB, Willson TM, Zetterstrm RH, et al. An orphan nuclear receptor activated by pregnanes defines a novel steroid signaling pathway. Cell. 1998; 92: 73 Horwitz KB, Horwitz LD. Canine vascular tissues are targets for androgens, estrogens, progestins, and glucocorticoids. J Clin Invest. 1982; 69: 750 Lin AL, Gonzalez R, Carey KD, Shain SA. Estradiol-17 affects estrogen receptor distribution and elevates progesterone receptor content in baboon aorta. Arteriosclerosis. 1986; 6: 495504. Ingegno MD, Money SR, Thelmo W, Greene GL, Davidson M, Jaffe BM, Pertschuk LP. Progesterone receptors in the human heart and great vessels. Lab Invest. 1988; 59: 353356. Elices MJ, Osborn L, Takada Y, Crouse C, Luhowskyj S, Hemler ME, Lobb RR. VCAM-1 on activated endothelium interacts with the leukocyte integrin VLA-4 at a site distinct from the VLA-4 fibronectin binding site. Cell. 1990; 60: 577584. Otsuki M, Hashimoto K, Morimoto Y, Kishimoto T, Kasayama S. Circulating vascular cell adhesion molecule-1 VCAM-1 ; in atherosclerotic NIDDM patients. Diabetes. 1997; 46: 2096 Rohde LE, Lee RT, Rivero J, Jamacochian M, Arroyo LH, Briggs W, Rifai N, Libby P, Creager MA, Ridker PM. Circulating cell adhesion molecules are correlated with ultrasound-based assessment of carotid atherosclerosis. Arterioscler Thromb Vasc Biol. 1998; 18: 17651770. Jensen J, Riis BJ, Strom V, Nilas L, Christiansen C. Long-term effects of percutaneous estrogens, and progesterone on serum lipoproteins in postmenopausal women. J Obstet Gynecol. 1987; 156: 66 Miler VT, Muesing RA, Larosa JC. Effects of conjugated equine estrogen with and without three different progestogens on lipoproteins, highdensity lipoprotein subtractions, and apolipoprotein A-1. Obstet Gynecol. 1991; 77: 235240. Lobo RA. Effects of hormonal replacement on lipids and lipoproteins in postmenopausal women. J Clin Endocrinol Metab. 1991; 73: 925930. Levine RL, Chen S-J, Durand J, Chen Y-F, Oparil S. Medroxyprogesterone attenuates estrogen-mediated inhibition of neointima formation after balloon injury of the rat carotid artery. Circulation. 1996; 94: 22212227. Adams MR, Register TC, Golden DL, Wagner JD, Williams JK. Medroxyprogesterone acetate antagonized inhibitory effects of conjugated equine estrogens on coronary artery atherosclerosis. Arterioscler Thromb Vasc Biol. 1997; 17: 217221. McKenna NJ, Lanz RB, O'Malley BW. Nuclear receptor coregulators: cellular and molecular biology. Endocr Rev. 1999; 20: 321344. Perkins ND, Felzien LK, Betts JC, Leung K, Beach DH, Nabel GJ. Regulation of NF- B by cyclin-dependent kinases associated with the p300 coactivator. Science. 1997; 275: 523527. Gerritsen ME, Williams AJ, Neish AS, Moore S, Shi Y, Collins T. CREB-binding protein p300 are transcriptional coactivators of p65. Proc Natl Acad Sci U S A. 1997; 94: 29272932. Na SY, Lee SK, Han SJ, Choi HS, Im SY, Lee JW. Steroid receptor coactivator-1 interacts with the p50 subunit and coactivates nuclear factor B-mediated transactivations. J Biol Chem. 1998; 273: 1083110834. Kalkhoven E, Wissink S, van der Saag PT, van der Burg B. Negative interaction between the RelA p65 ; subunit of NF- B and the progesterone receptor. J Biol Chem. 1996; 271: 6217 McKay LI, Cidlowski JA. Cross-talk between nuclear factor- B and the steroid hormone receptors: mechanisms of mutual antagonism. Mol Endocrinol. 1998; 12: 4556 and flagyl.

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Estradiol Eztradiol Estrogel Estrasorb 1.25 grams 3.5 grams Once daily Once daily $40 to $49 $47 to $62. Hallucinogenics". Tr. 5717-18 ; . She stated, however, that she may have been present at the murders, explaining that in her dreams she had visions of Mrs. MacDonald's bed with the word "pig" written in blood on the headboard, and of herself on a couch in the MacDonald home with MacDonald holding a bloody icepick Tr. 5719-20 ; . But she immediately retracted the statement, explaining that she only thought she heard of MacDonald before the murders Tr. 5718 ; . Finally, she informed Brisentine that she knew the identity of the persons who murdered the MacDonald family and that she would furnish the identity of the murderers in exchange for immunity Tr. 5721 ; . The following day, Stoeckley again recanted her statement concerning participation in the murders. Asserting that she had "said too much, " she admitted that she had been lying when she told Bristentine she knew the identities of the killers; in fact, she only suspected some people of committing the homicides Tr. 5721-22 ; . During her stay in Nashville, Stoeckley made statements to several acquaintances concerning her possible involvement in the murders of the MacDonald family. In November 1970, while ill with hepatitis, "incoherent", and in a state of hysteria, she told Jane Zillioux, a neighbor, that she could not return to her home in Fayetteville, North Carolina because she had been involved in some murders with three boys whose identities she did not know. When Zillioux pressed her for additional details, Stoeckley admitted that she did not know whether or not she had participated in the murders, explaining, "I've been a heavy drug user and . drugs make you and fluconazole.

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And 6 ; , 150 mg patients 7-9 ; , and 250 mg monthly patients 10-13 ; . Despite the normalization of PRL levels with 50 mg dose, subject 2 received a higher dose 100 mg ; , as no change in tumor size was found after 6 months of therapy Table 2 ; . Plasma PRL levels were measured 8, 12, 16, and 20 h before the first injection; on days 1, 14, and 28 after each injection during the first year; and on days 1 and 28 after each injection thereafter. All patients had nuclear magnetic resonance imaging MRI ; scans Ansaldo RM Esatom Esaote 0.5 Tesla; subjects 1, 2, and 5-13 ; or high resolution computed tomography CT ; with contrast enhancement General Electric CT T 8800; patients 3 and 4 ; before and 1, 6, 12, and 48 months after therapy. Occasionally, a CT scan was also performed in the other patients. Before the study, at yearly intervals during treatment, and up to the end of the observation period, all patients had TJ, Tl, free T free T4, TSH, cortisol, FSH, LH, estradiol, or testosterone evaluations as well as electrocardiogram and routine toxicological screen of hemoglobin, white count, plasma urea, creatinine, electrolytes, liver enzymes, bilirubin, calcium, and phosphate. Plasma PRL levels were measured by immunoradiometric assay, using reagents PRL-CTK-IRMA ; provided by Sorin Biomedica Saluggia, Italy the intra- and interassay coefficients of variations were 3.2% and 6.9%, respectively. Normal PRL values were 3-20 fig L in females and 3-12 rg L in males. Plasma testosterone levels were measured by RIA, using reagents DIRIA-TESTOK ; provided by Sorin Biomedica; intraand interassay coefficients of variations were 7.5% and 8.2%, respec and galantamine. Fig. 3: Relative mRNA expression of luteinizing hormone -subunit LH ; , follicle-stimulating hormone -subunit FSH ; and gonadotropin releasing hormone GnRH ; expression of male X. laevis in response to anti ; estrogenic, anti ; androgenic model compounds and to Lambro river water. Ethinylestradiol EE2 ; , tamoxifen TAM ; , methyldihydrotestosterone MDHT ; and flutamide FLU ; were used as estrogenic, antiestrogenic, androgenic and antiandrogenic compound, respectively, at a concentration of 10 -8 M. refers to the solvent control and Lam to Lambro river water. Results were expressed relative to control values as means SD, normalized by the house-keeping gene elongation factor 1-alpha EF 1- ; . Means were derived from samples of 8 individuals each. Significant differences between treatments and control groups were tested by ANOVA followed by Dunnetts test. Significant differences were indicated by asterisks * p 0.05, * p 0.01, * p 0.001. Ian hamster. For this purpose, estdadiol and progesterone metabolites were quantified by solid phase RIA in feces of intact, sexually mature female hamsters during an estrous cycle. In order to determine if fecal steroid concentrations reflect changes in ovarian status, estrogen and progesterone metabolites were also quantified in superovulated female feces and glibenclamide.
Potential New Therapies for Lupus Nephritis Continued ; depressive disorder, seizures, and migraine headaches ; was given to mice with lupus-like kidney disease and was found to reduce the severity of kidney disease symptoms. Other studies with lupus mouse models suggest that mithramycin, an antibiotic used to treat tumors and trichostatin A TSA ; might be potential treatments for lupus-like kidney disease. Clinical trials on people with lupus would need to be performed to determine the value of these agents. Kidney disease can be difficult to diagnose as it sometimes does not have many signs or symptoms. Symptoms can include: weight gain, dark or pink or foamy urine, swelling edema ; of the legs, eyelids and hands, and increased fatigue or tiredness. However, other illnesses may have similar symptoms. Lupus is a chronic autoimmune disease that causes inflammation and tissue damage to virtually any organ system in the boby. Common symptoms of lupus include achy or swollen joints, extreme exhaustion, fevers, and skin rashes, especially on the face, scalp and neck. Approximately 1.5 million Americans have a form of lupus, 90 percent of whom are women. Lupus is more common among people of color. Lupus occurs two to three times more often among African Americans, Asians, Hispanics and Native Americans than among Caucasians, a health disparity that reamins unexplained. The Lupus Foundation of America is the nation's leading nonprofit voluntary health agency dedicated to finding the causes and cure for lupus. For more information about lupus and lupus nephritis, visit the LFA website at lupus or call 1888-38-LUPUS, or your local chapter, to request a free brochure. Combining prescription medicines and otc medicines can lead to problematic drug interactions and glucovance and estradiol, because estradilo hemihydrate. Table 2 - Serum prolactin levels of intact or ovariectomized rats treated with etradiol valerate alone or in combination with bromocriptine. Results are reported as mean SEM. The number of animals is given in parentheses. BCP, Bromocriptine. aP 0.05 versus intact rats Duncans test * P 0.05 versus estradiol t-test ; . Estraadiol dose rat-1 week-1 ; Intact rats 50 g, 2 weeks 50 g, 4 weeks 50 g, 10 weeks 300 g, 2 weeks 300 g, 4 weeks 300 g, 10 weeks Prolactin levels ng ml ; 9.11 39.70 80.10 ; 3.60a 5 ; 18.3a 5 ; 23.6a 8 ; 96.1a 9 ; 51.0a 8 ; 22.0a 8 ; Estrasiol + BCP dose BCP rat -1 day-1 ; Prolactin levels ng ml.

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To demonstrate how Dynabeads MyOneTM Immunoassay Carboxyl ; improve antigenbased assays, the beads were tested in model systems using a peptide insulin ; and a steroid estradiol ; antigen. The data presented are from model systems for testing bead functionality and do not reflect the results of an optimized clinical assay and inderal.

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ST step therapy; QL quantity limits * Step therapy requires certain first-line drugs: generic drugs tier 1 ; or preferred brand drugs tier 2 ; , to be prescribed prior to approval of specific second-line drugs: preferred brand drugs tier 2 ; or non-preferred brand drugs tier 3 ; . First-line therapy includes generic drugs and some preferred brand drugs on the Preferred Drug List PDL ; . Certain prescription drugs may be prescribed only in limited quantities. Drugs that have quantity limits are identified on the PDL with after the drug name. Phosphorylation and reduced expression and tyrosine phosphorylation of insulin receptor substrate 1 IRS-1 ; .24 Prolonged exposure of adipocytes to increased plasma estradiol, for example, during pregnancy25 or in women with central obesity, 26, 27 appears to be to prone to decreased IRS-1.
AGE-ADJUSTED RATE--Cancer incidence and mortality statistics are weighted age-adjusted ; to match the age distribution of the population during a given census year. This allows for comparison of rates between different regions of the country. Unless rates are age-adjusted, a region with more older people would have a higher rate of breast cancer than a region with younger population. AROMATIC AMINE--A pollutant from the chemical and plastics industries, and a byproduct of high temperature cooking of meat and fish. Many aromatic amines are known to cause mammary tumors in animals. CARCINOGEN--Any substance or process known to cause cancer. CASE-CONTROL STUDY--A research study that looks at two groups of people, one group that has a disease cases ; and one that does not have the disease controls ; but is otherwise similar in age, lifestyle and education to the cases. The goal is to help identify potential causes of the disease. For example, women who have breast cancer might be compared with women who do not have breast cancer to see whether use of pesticides at home was more common among cases than among controls. DIOXIN--The name given to a group of highly toxic chemicals created by industrial processes that use chlorine, such as the manufacture of paper or the incineration of polyvinyl chloride PVC ; plastics. DNA ADDUCT--A marker of exposure to a particular carcinogen such as those found in PAHs or tobacco smoke, creating an altered form of DNA. If normal repair mechanisms are successful, the DNA returns to its original structure. If mis-repaired, the adduct results in a mutation, increasing the risk of cancer. ELECTROMAGNETIC FIELDS EMFs ; --Nonionizing radiation that includes electrical fields, magnetic fields, radiofrequency transmissions and microwaves. ENDOCRINE DISRUPTING CHEMICALS COMPOUNDS EDCs ; --Chemicals such as dioxin that disturb the body's finely tuned hormonal endocrine ; balance. Any disruption in hormonal activity can interfere with an organism's ability to grow, develop and function normally. Some EDCs act like the hormone estrogen and may be referred to as xenoestrogens. EPIDEMIOLOGY--The study of the distribution and determinants of disease frequency in human populations. EPITHELIAL CELLS--Most internal and external surfaces of the body are covered with epithelial tissue, including the ducts and lobules of the breast. The majority of breast cancers originate in the epithelial cells of the ducts and are called ductal carcinoma. ESTRADIOL--The most potent of the three principal types of estrogen hormones; the other types are estriol and estrone. Estadiol is produced by the ovaries, the cortex outer layer ; of the adrenal glands and, during pregnancy, by the placenta. Rstradiol is also produced by the male testes. ESTRIOL--A weaker estrogen produced in the ovaries and peripheral body fat when androgens male hormones ; are converted to estrone by the action of aromatase. Estriol is the metabolite of estrone and estradiol. ESTROGEN-DEPENDENT--Tumors that grow in the presence of natural or synthetic estrogen are said to be estrogen-dependent or estrogenreceptor-positive see next page ; . Tamoxifen is often used to treat such tumors because it reduces estrogen levels in the breast. Below you will find a year's worth of ePharm5 briefs about DTC, education-to-consumer ETC ; , and educationto-physician news published in our daily e-zine. If you are an InDepth subscriber, you have access to these archives by conducting a search on the InDepth site. December 15, 2005 Pharmas can support Part D education with unbiased funding Opportunities exist for pharmas to help educate beneficiaries about Medicare Part D by providing assistance to managed care Part D plan sponsors, according to a survey of managed care organizations and PBMs. Nearly half of the respondents said they may be willing to accept some manufacturer support, although they were careful to stipulate that assistance must be general and brand-neutral, according to the study by GfK Market Measures, formerly NOP World Health. GfK Market Measures says the results suggest that unrestricted educational grants or support of public information campaigns would be the best ways for pharmas to assist managed care. In June, Verispan reported that Eli Lilly, Merck, and Pfizer had been the most active pharmas in reaching out to managed care organizations' Part D issues ePharm5, 6 10 05 ; . December 12, 2005 Manhattan Research: Integrate `e' into physician marketing mix Fully integrating the e-channel into physician marketing and segmenting and targeting consumer behavior emerged as key topics during Manhattan Research's new Opinion Leader Roundtable. "I would look to physician portals as something that will become a must-have for pharma in the next year to two years in the states, " AstraZeneca's John Fish said. He said that pharmas' e-marketing and sales activities should be better coordinated. The panelists also said the Internet will play an important role in moving away from mass DTC and toward relationship marketing. Because pharmas can leverage the online channel to deliver targeted and more substantive information to consumers, it has become a more effective and measurable way to reach patients, Forest Lab's Claire Zinnes said. To see all of Manhattan Research's Opinion Leader Roundtable, featuring discussion with panelists from AstraZeneca, Roche Labs, Genentech, and Forest Labs, go to ePharm InDepth and look under "White Papers and Reports." December 9, 2005 Manhattan Research: IT has major role in European physician marketing With 65% of practicing European physicians saying that Internet access is critical to their practice and 86% saying they have Internet access in their offices, IT plays a significant role for doctors across Europe, according to a new study from Manhattan Research. Despite the doctors' use of the Internet, many pharmas still view Europe as several years behind the United States, Mark Bard, Manhattan Research president, tells ePharm5. "The latest research clearly reveals an opportunity for pharmaceutical companies to explore opportunities in the networked economy on a global basis, " he says, adding that pharmas should develop country-specific strategies. He also says that although the European market tends to lag in areas such as pharma promotional activities online and famotidine. 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